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This response targets dysfunctional organelles for clearance and activates the AMPK-ULK1-Beclin-1 signaling axis, which initiates autophagosome formation

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However, to get further insight in the role of SLC35A39 in the brain, the generation of specific conditional Slc25a39 knockout mice that carry a deletion of Slc25a39 exclusively in neurons or in the central nervous system may further boost our understanding on the roles and in the importance of SLC25A39 in neuronal tissues

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